Ja imam čak nekoliko slučajeva u bližoj okolini i široj porodici koji su slični tvom. Uglavnom momci tvojih godina koji iznenada osete promene srčanog ritma. Uglavnom dolazi do naglog smanjenja broja otkucaja srca. Od mog prijatelja sin je čak došao do momenta kada se odlučivalo o hitnoj ugradnji pejsmejkera. Na svu sreću nije to uradio. Danas ima 25 god i nema više nikakvih problema.
Drugi momak, sin drugarice moje supruge, nije izdržao pritisak i strah, ugradili su mu aparat u 19-toj. Od bratovog šuraka sin je u istim godinama imao problem sa srcem. Sumnjali su na Koksaki virus. Danas je oženjen, ima 26 godina i nema nikakvih problema sa srcem. Sin jednog kolege sa posla je takođe imao isti problem. Takođe su sumnjali na virus. Sve je prošlo samo od sebe.
Mislim da je ova pojava vezana za odrastanje i hormone. Verovatno u tim godinama dostižete maksimum Testosterona koji se verovatno transformiše i u Dihidrotestosteron.
Ovi androgeni hormoni imaju direktan uticaj na program rada srčanog mišića.
"Most of the research concerning the effects of gonadal hormones on the cardiovascular reflexes has focused on 17p-estradiol. However, other studies have provided evidence that androgens (including testosterone) play an important role in the control of cardiovascular function by modulation of cardiovascular reflexes (Caminiti et al., 2009). Steroids can cross the blood-brain barrier and act on the central nervous system, where androgen receptors in the central cardiovascular regulatory regions, such as NA and DMNX (Peuler et al., 1990; Pouliot et al., 1996) have been demonstrated. Therefore it is possible that androgens may act on brainstem vagal preganglionic neurons to modulate cardiomotor vagal activity. In accordance with this data, El-Mass et al. (2001) have shown that in male rats, castration caused a significant attenuation of baroreceptor control of reflex bradycardia versus no effect on reflex tachycardia. Testosterone replacement increased BRS to phenylephrine in castrated rats and restored reflex bradycardic responses to levels similar to those of sham-operated rats. The muscarinic blockade by atropine in sham-operated rats caused a substantial reduction in BRS to phenylephrine, an effect that was significantly attenuated by castration and restored to sham-operated levels after testosterone replacement, suggesting that testosterone facilitates baroreceptor control of reflex bradycardia. Moreover, the modulatory role of testosterone on baroreflex responsiveness appears to involve, at least partly, enhancement of cardiac vagal efferent activity. Corroborating these data, a long-term testosterone therapy (6 weeks) improves the baroreflex sensitivity in men with chronic heart failure (Caminiti et al., 2009). The blockade of androgen receptor with flutamide attenuates the enhancement of baroreflex bradycardia in sexually mature male rats, indicating that the effects of testosterone on BRS depend on the involvement of the androgen receptor (Ward and Abdel-Rahman, 2006).
Besides the testosterone-induced effects on baroreflex, this sexual hormone may also modulate the cardiopulmonary reflex and the chemoreflex. Bissoli et al (2009) demonstrated that long-term treatment (8 weeks) with supraphysiological doses of nandrolone decanoate reduces the sensitivity of BJR control of HR in male rats. The effects of testosterone on BJR seem to be time-dependent, since the same treatment for 4 weeks had no effects on BJR nor the basal HR (Andrade et al., 2008). Pereira-Junior et al. (2006) showed that 10 weeks of high-dose nandrolone decanoate treatment leads to dysfunction in tonic cardiac autonomic regulation, with marked impairment of parasympathetic cardiac modulation and sympathetic hyperactivity. Regarding the chemoreflex, data from castrated male cats suggest that testosterone increases the hypoxic and hypercapnic ventilatory responses and augmented carotid body sensitivity to hypoxia (Behan et al., 2003). In adult rats, however, castration had no effect on the ventilatory response measured at the end of hypoxia (Joseph et al., 2002). On the other hand, Bairam et al. (2009) demonstrated that gonadectomy increased the acute breathing frequency response to hypoxia in neonatal rats. Because the rapid increase in breathing frequency is attributed to peripheral chemoreceptor activation, these data suggest that testosterone attenuates carotid body function. Although several studies demonstrated contradictory results about the benefic or malefic effects of testosterone on the modulation of cardiovascular reflexes, the characterization of the mechanisms could lead to a better understanding of the effects of testosterone in cardiovascular system and to the development of new therapies."
Nemoj da paničiš sinak, to ti je najvažnije. To je samo faza koja će proći sama od sebe. Nabavi Epilobium angustifolium, to je biljka koja ima antiandrogeno svojstvo. Smiriće ti hormone a i anksioznost uz to.
Nemoj da obraćaš toliku pažnju na srce, trudi se da ga ne primećuješ, ne meri puls tako često, i ono će se samo smiriti. Verovatno si uspostavio psihosomatski feedback, kog nisi ni svestan, a koji ne umeš da prekineš. Jedan dobar psihoterapeut bi te vrlo brzo vratio u sedlo veruj mi.
Ne očekuj puno od hemije sinak, nije to za tebe. Prvo što nema razloga da se truješ lekovima koji izazivaju zavisnost, a drugo oni ti neće povratiti stvarni mir, samo će zamaskirati problem, i verovatno još zakomplikovati problem sa srčanim ritmom.
Seroksat deluje na promenu srčanog ritma, a o ksalolu već znaš da :
"Obratite se lekaru ako se tokom primene Ksalola pojave neke od sledećih nuspojava: nemir, izbudjenost, izlivi agresije, bes, ljutnja, razdražljivost, pogrešna mišljenja i uverenja , halucinacije, gubitak pamćenja ili žutica.
Ostale moguće nuspojave: pospanost, umor, slabost u mišićima, otežan govor, zamagljen vid, loša koordinacija pokreta, manjak pažnje, koncentracije, promena telesne težine, mučnina, povraćanje, dijareja, bol u stomaku, suva usta, osipi, teškoće sa mokrenjem, poremećeni menstrualni ciklus, jači ili pak oslabljeni lobido."
